Abstract

This article presents the case of a young woman with a case of Wernicke’s encephalopathy and polyneuropathy associated with Beri-Beri, vitamin B deficiency and we focus on the electroneurographic and electromyographic description of the case and its differences with the AIDP. Guillen Barre syndrome. In this patient, the presence of an axonal involvement, not demyelinating, of sensory and motor nerves of predominance in MMII of asymmetric distribution and acute course was clearly verified. If polyneuropathy is associated with encephalopathy, the picture known as Wernicke’s syndrome and polyneuropathy due to thiamine deficiency occurs [2,3,5,6]. This patient was seen by us in January and then in October 8 months later ostensibly improving sensory and motor neurography.

His article presents the case of a young woman with a case of Wernicke encephalopathy and polyneuropathy associated with beriberi, vitamin B deficiency, and we focus on the electroneurographic and electromyographic description of the case and its differences with the AIDP. Guillén Barre syndrome. In this patient, the presence of non-demyelinating axonal involvement of sensory and motor nerves, predominantly in MMII of asymmetric distribution and of acute course, was clearly verified. If the polyneuropathy is associated with encephalopathy, the condition known as Wernicke syndrome and thiamine deficiency polyneuropathy occurs. This patient was seen 8 months later with marked improvement in sensory and motor neurography.

Keywords: Polyneuropathy; Beri-Beri; Thiamine Deficiency; Axonal Lesion; EMG; TIQA MINA; PYRIDOXINE

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