Editorial
Volume 4 Issue 9 - 2020
Hyperglycemia and Covid-19: State of the Art
1Department of Internal Medicine, Ospedale Cav. R. Apicella - ASL Napoli 3 Sud, Via di Massa, Pollena, Napoli, Italy
2Covid Hospital Boscotrecase - ASL Napoli 3 Sud, Via Lenza, Boscotrecase, Napoli, Italy
*Corresponding Author: Carmine Finelli, Department of Internal Medicine, Ospedale Cav. R. Apicella - ASL Napoli 3 Sud, Via di Massa,
Pollena, Napoli, Italy.
Received: August 25, 2020; Published: August 29, 2020
Coronavirus disease infection-2019 (COVID-19) poses a twofold danger of patients with diabetes. Diabetes has been reported as a contributing factor for the seriousness of the condition, but in cases where food intake is lower and more nuanced, patients prefer to regulate glucose [1]. COVID-19 is caused by coronavirus SARS-CoV-2 (severe acute respiratory syndrome coronavirus-2), which has spread exponentially in several countries across the world [2,3]. Older patients and individuals of all ages with severe underlying health problems in conjunction with obesity could be at elevated risk of COVID-19 acute disease. While general obesity is a risk factor for several diseases, many clinical trials have shown that visceral fat accumulation is most strongly linked to specific health conditions, such as cardiovascular disorders, insulin resistance and type 2 diabetes mellitus [3-5]. In addition, effective cardiometabolic monitoring of patients suffering from extreme COVID-19 disease could be appropriate [6].
References
- Finelli C. “Diabetes and Covid 19: Current Status”. EC Diabetes and Metabolic Research 7 (2020): 09-12.
- Ge H., et al. “The epidemiology and clinical information about COVID-19”. European Journal of Clinical Microbiology and Infectious Diseases 6 (2020): 1011-1019.
- Finelli C. “Obesity and Covid 19: Possible Risks”. EC Endocrinology and Metabolic Research 6 (2020): 01-03.
- Finelli C., et al. “Should visceral fat be reduced to increase longevity?” Ageing Research Reviews 4 (2013): 996-1004.
- Finelli C. “Metabolic Syndrome and Adipose Tissue: Potential Connections”. EC Endocrinology and Metabolic Research 9 (2019): 35-37.
- Finelli C. “Metabolic Syndrome and COVID 19: Current Condition”. EC Endocrinology and Metabolic Research 7 (2020): 01-02.
- Finelli C. “Obesity, COVID-19 and immunotherapy: the complex relationship!”. Immunotherapy (2020);10.2217/imt-2020-0178. doi:10.2217/imt-2020-0178.
- Samavati L and Uhal BD. “ACE2, Much More Than Just a Receptor for SARS-COV-2”. Frontiers in Cellular and Infection Microbiology 10 (2020): 317.
- Hussain A., et al. “COVID-19 and diabetes: Knowledge in progress”. Diabetes Research and Clinical Practice 162 (2020): 108142.
- Wang S., et al. “Fasting blood glucose at admission is an independent predictor for 28-day mortality in patients with COVID-19 without previous diagnosis of diabetes: a multi-centre retrospective study”. Diabetologia (2020): 1-10.
- Yang JK., et al. “Binding of SARS coronavirus to its receptor damages islets and causes acute diabetes”. Acta Diabetologica 3 (2020): 193-199.
- Perrotta F., et al. “Severe respiratory SARS-CoV2 infection: Does ACE2 receptor matter?” Respiratory Medicine 168 (2020): 105996.
- Somasundaram NP., et al. “The Impact of SARS-Cov-2 Virus Infection on the Endocrine System”. Journal of the Endocrine Society 8 (2020): bvaa082.
- Leung JM., et al. “ACE-2 expression in the small airway epithelia of smokers and COPD patients: implications for COVID-19”. European Respiratory Journal 5 (2020): 2000688.
- Belouzard S., et al. “Mechanisms of coronavirus cell entry mediated by the viral spike protein”. Viruses 6 (2012): 1011-1033.
Citation: Carmine Finelli. “Hyperglycemia and Covid-19: State of the Art". EC Diabetes and Metabolic Research 4.9 (2020): 28-29.
Copyright: © 2020 Carmine Finelli. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
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